Inflammation og frie oxygenradikaler

Publikation: Bidrag til tidsskrift › Tidsskriftartikel › Forskning › fagfællebedømt

Standard

Inflammation og frie oxygenradikaler. / Nielsen, O H; Ahnfelt-Rønne, I.

I: Nordisk Medicin, Bind 104, Nr. 8-9, 1989, s. 216-8.

Publikation: Bidrag til tidsskrift › Tidsskriftartikel › Forskning › fagfællebedømt

Harvard

Nielsen, OH & Ahnfelt-Rønne, I 1989, 'Inflammation og frie oxygenradikaler', Nordisk Medicin, bind 104, nr. 8-9, s. 216-8.

APA

Nielsen, O. H., & Ahnfelt-Rønne, I. (1989). Inflammation og frie oxygenradikaler. Nordisk Medicin, 104(8-9), 216-8.

Vancouver

Nielsen OH, Ahnfelt-Rønne I. Inflammation og frie oxygenradikaler. Nordisk Medicin. 1989;104(8-9):216-8.

Author

Nielsen, O H ; Ahnfelt-Rønne, I. / Inflammation og frie oxygenradikaler. I: Nordisk Medicin. 1989 ; Bind 104, Nr. 8-9. s. 216-8.

Bibtex

@article{5ba719c7ac9b4b0baa8ba0933436064d,
title = "Inflammation og frie oxygenradikaler",
abstract = "When mammalian phagocytes encounter appropriate stimuli, highly reactive oxygen derived free radicals (superoxide and hydroxyl) and related species (hydrogen peroxide and hypochlorous acid) may be produced as a normal mechanism in the defence of the host against invading microorganisms. The radicals are destructive to most biological molecules and are responsible for much of the damage inflicted by phagocytes on both microorganisms and surrounding tissues at sites of infection or inflammation. Increasing evidence suggests that the generation of oxygen metabolites plays an important role not only for perpetuation of chronic inflammation, but also for the etiology of other health problems i.e. ageing, cancer, ischemia, and radiationinduced injuries. The extent of tissue damage in inflammation may partly be the result of the balance between free radicals generated and the antioxidant/radical scavenger protective defence systems. The implication of oxygen free radicals in inflammation and future directions are briefly discussed in the present overview.",
keywords = "Free Radicals, Humans, Inflammation/immunology, Phagocytes/immunology",
author = "Nielsen, {O H} and I Ahnfelt-R{\o}nne",
year = "1989",
language = "Dansk",
volume = "104",
pages = "216--8",
journal = "Nordisk medicin",
issn = "0029-1420",
publisher = "L{\ae}geforeningens Forlag",
number = "8-9",

}

RIS

TY - JOUR

T1 - Inflammation og frie oxygenradikaler

AU - Nielsen, O H

AU - Ahnfelt-Rønne, I

PY - 1989

Y1 - 1989

N2 - When mammalian phagocytes encounter appropriate stimuli, highly reactive oxygen derived free radicals (superoxide and hydroxyl) and related species (hydrogen peroxide and hypochlorous acid) may be produced as a normal mechanism in the defence of the host against invading microorganisms. The radicals are destructive to most biological molecules and are responsible for much of the damage inflicted by phagocytes on both microorganisms and surrounding tissues at sites of infection or inflammation. Increasing evidence suggests that the generation of oxygen metabolites plays an important role not only for perpetuation of chronic inflammation, but also for the etiology of other health problems i.e. ageing, cancer, ischemia, and radiationinduced injuries. The extent of tissue damage in inflammation may partly be the result of the balance between free radicals generated and the antioxidant/radical scavenger protective defence systems. The implication of oxygen free radicals in inflammation and future directions are briefly discussed in the present overview.

AB - When mammalian phagocytes encounter appropriate stimuli, highly reactive oxygen derived free radicals (superoxide and hydroxyl) and related species (hydrogen peroxide and hypochlorous acid) may be produced as a normal mechanism in the defence of the host against invading microorganisms. The radicals are destructive to most biological molecules and are responsible for much of the damage inflicted by phagocytes on both microorganisms and surrounding tissues at sites of infection or inflammation. Increasing evidence suggests that the generation of oxygen metabolites plays an important role not only for perpetuation of chronic inflammation, but also for the etiology of other health problems i.e. ageing, cancer, ischemia, and radiationinduced injuries. The extent of tissue damage in inflammation may partly be the result of the balance between free radicals generated and the antioxidant/radical scavenger protective defence systems. The implication of oxygen free radicals in inflammation and future directions are briefly discussed in the present overview.

KW - Free Radicals

KW - Humans

KW - Inflammation/immunology

KW - Phagocytes/immunology

M3 - Tidsskriftartikel

C2 - 2780275

VL - 104

SP - 216

EP - 218

JO - Nordisk medicin

JF - Nordisk medicin

SN - 0029-1420

IS - 8-9

ER -

ID: 218728938