Michal Tomasz Marzec

Michal Tomasz Marzec

Gæsteforsker

ORCID: 0000-0001-9698-6019

Udgivelsesår:
Alle
1 - 3 ud af 3Pr. side: 10
Sortering: Udgivelsesdato
  1. 2005

  2. Activation of Mammalian Target of Rapamycin in Transformed B Lymphocytes Is Nutrient Dependent but Independent of Akt, Mitogen-Activated Protein Kinase/Extracellular Signal-Regulated Kinase Kinase, Insulin Growth Factor-I, and Serum.

    Wlodarski, P., Kasprzycka, M., Liu, X., Marzec, Michal Tomasz, Robertson, E. S., Slupianek, A. & Wasik, M. A., 2005, I: Cancer Research. 65, 17, s. 7800-7808 9 s.

    Publikation: Bidrag til tidsskrift › Tidsskriftartikel › fagfællebedømt

  3. Udgivet

    Inhibition of ALK enzymatic activity in T-cell lymphoma cells induces apoptosis and suppresses proliferation and STAT3 phosphorylation independently of Jak3

    Marzec, Michal Tomasz, Kasprzycka, M., Ptasznik, A., Wlodarski, P., Zhang, Q., Ødum, Niels & Wasik, M. A., 2005, I: Laboratory Investigation. 85, 12, s. 1544-54 10 s.

    Publikation: Bidrag til tidsskrift › Tidsskriftartikel › fagfællebedømt

  4. STAT3- and DNA methyltransferase 1-mediated epigenetic silencing of SHP-1 tyrosine phosphatase tumor suppressor gene in malignant T lymphocytes.

    Zhang, Q., Wang, H. Y., Marzec, Michal Tomasz, Raghunath, P. N., Nagasawa, T. & Wasik, M. A., 2005, I: Proceedings of the National Academy of Sciences of the United States of America. 102, 19, s. 6948-6953 6 s.

    Publikation: Bidrag til tidsskrift › Tidsskriftartikel › fagfællebedømt

ID: 129941679