Continuous parenteral and enteral nutrition induces metabolic dysfunction in neonatal pigs

Research output: Contribution to journalJournal articlepeer-review

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Continuous parenteral and enteral nutrition induces metabolic dysfunction in neonatal pigs. / Stoll, Barbara; Puiman, Patrycja Jolanta; Cui, Liwei; Chang, Xiaoyan; Benight, Nancy Marie; Bauchart-Thevret, Caroline; Hartmann, Bolette; Holst, Jens Juul; Burrin, Douglas Guy.

In: Journal of Parenteral and Enteral Nutrition, Vol. 36, No. 5, 09.2012, p. 538-50.

Research output: Contribution to journalJournal articlepeer-review

Harvard

Stoll, B, Puiman, PJ, Cui, L, Chang, X, Benight, NM, Bauchart-Thevret, C, Hartmann, B, Holst, JJ & Burrin, DG 2012, 'Continuous parenteral and enteral nutrition induces metabolic dysfunction in neonatal pigs', Journal of Parenteral and Enteral Nutrition, vol. 36, no. 5, pp. 538-50. https://doi.org/10.1177/0148607112444756

APA

Stoll, B., Puiman, P. J., Cui, L., Chang, X., Benight, N. M., Bauchart-Thevret, C., Hartmann, B., Holst, J. J., & Burrin, D. G. (2012). Continuous parenteral and enteral nutrition induces metabolic dysfunction in neonatal pigs. Journal of Parenteral and Enteral Nutrition, 36(5), 538-50. https://doi.org/10.1177/0148607112444756

Vancouver

Stoll B, Puiman PJ, Cui L, Chang X, Benight NM, Bauchart-Thevret C et al. Continuous parenteral and enteral nutrition induces metabolic dysfunction in neonatal pigs. Journal of Parenteral and Enteral Nutrition. 2012 Sep;36(5):538-50. https://doi.org/10.1177/0148607112444756

Author

Stoll, Barbara ; Puiman, Patrycja Jolanta ; Cui, Liwei ; Chang, Xiaoyan ; Benight, Nancy Marie ; Bauchart-Thevret, Caroline ; Hartmann, Bolette ; Holst, Jens Juul ; Burrin, Douglas Guy. / Continuous parenteral and enteral nutrition induces metabolic dysfunction in neonatal pigs. In: Journal of Parenteral and Enteral Nutrition. 2012 ; Vol. 36, No. 5. pp. 538-50.

Bibtex

@article{0537d72261a94d129e1d77f6419b8fda,
title = "Continuous parenteral and enteral nutrition induces metabolic dysfunction in neonatal pigs",
abstract = "We previously showed that parenteral nutrition (PN) compared with formula feeding results in hepatic insulin resistance and steatosis in neonatal pigs. The current aim was to test whether the route of feeding (intravenous [IV] vs enteral) rather than other feeding modalities (diet, pattern) had contributed to the outcome.",
keywords = "Administration, Intravenous, Animals, Animals, Newborn, Blood Glucose, Endpoint Determination, Enteral Nutrition, Fatty Liver, Female, Food, Formulated, Glucagon-Like Peptide 1, Glucagon-Like Peptide 2, Incretins, Inflammation, Insulin, Insulin Resistance, Intestine, Small, Liver, Metabolic Diseases, Nonlinear Dynamics, Organ Size, Parenteral Nutrition, Swine",
author = "Barbara Stoll and Puiman, {Patrycja Jolanta} and Liwei Cui and Xiaoyan Chang and Benight, {Nancy Marie} and Caroline Bauchart-Thevret and Bolette Hartmann and Holst, {Jens Juul} and Burrin, {Douglas Guy}",
year = "2012",
month = sep,
doi = "10.1177/0148607112444756",
language = "English",
volume = "36",
pages = "538--50",
journal = "Journal of Parenteral and Enteral Nutrition",
issn = "0148-6071",
publisher = "SAGE Publications",
number = "5",

}

RIS

TY - JOUR

T1 - Continuous parenteral and enteral nutrition induces metabolic dysfunction in neonatal pigs

AU - Stoll, Barbara

AU - Puiman, Patrycja Jolanta

AU - Cui, Liwei

AU - Chang, Xiaoyan

AU - Benight, Nancy Marie

AU - Bauchart-Thevret, Caroline

AU - Hartmann, Bolette

AU - Holst, Jens Juul

AU - Burrin, Douglas Guy

PY - 2012/9

Y1 - 2012/9

N2 - We previously showed that parenteral nutrition (PN) compared with formula feeding results in hepatic insulin resistance and steatosis in neonatal pigs. The current aim was to test whether the route of feeding (intravenous [IV] vs enteral) rather than other feeding modalities (diet, pattern) had contributed to the outcome.

AB - We previously showed that parenteral nutrition (PN) compared with formula feeding results in hepatic insulin resistance and steatosis in neonatal pigs. The current aim was to test whether the route of feeding (intravenous [IV] vs enteral) rather than other feeding modalities (diet, pattern) had contributed to the outcome.

KW - Administration, Intravenous

KW - Animals

KW - Animals, Newborn

KW - Blood Glucose

KW - Endpoint Determination

KW - Enteral Nutrition

KW - Fatty Liver

KW - Female

KW - Food, Formulated

KW - Glucagon-Like Peptide 1

KW - Glucagon-Like Peptide 2

KW - Incretins

KW - Inflammation

KW - Insulin

KW - Insulin Resistance

KW - Intestine, Small

KW - Liver

KW - Metabolic Diseases

KW - Nonlinear Dynamics

KW - Organ Size

KW - Parenteral Nutrition

KW - Swine

U2 - 10.1177/0148607112444756

DO - 10.1177/0148607112444756

M3 - Journal article

C2 - 22549765

VL - 36

SP - 538

EP - 550

JO - Journal of Parenteral and Enteral Nutrition

JF - Journal of Parenteral and Enteral Nutrition

SN - 0148-6071

IS - 5

ER -

ID: 45841211