Arvelige hjertesygdomme forårsaget af natriumkanalmutationer

Research output: Contribution to journal › Journal article › Research › peer-review

Standard

Arvelige hjertesygdomme forårsaget af natriumkanalmutationer. / Tfelt-Hansen, Jacob; Olesen, Søren-Peter; Svendsen, Jesper Hastrup; Jespersen, Thomas.

In: Ugeskrift for læger, Vol. 171, No. 15, 2009, p. 1261-5.

Research output: Contribution to journal › Journal article › Research › peer-review

Harvard

Tfelt-Hansen, J, Olesen, S-P, Svendsen, JH & Jespersen, T 2009, 'Arvelige hjertesygdomme forårsaget af natriumkanalmutationer', Ugeskrift for læger, vol. 171, no. 15, pp. 1261-5.

APA

Tfelt-Hansen, J., Olesen, S-P., Svendsen, J. H., & Jespersen, T. (2009). Arvelige hjertesygdomme forårsaget af natriumkanalmutationer. Ugeskrift for læger, 171(15), 1261-5.

Vancouver

Tfelt-Hansen J, Olesen S-P, Svendsen JH, Jespersen T. Arvelige hjertesygdomme forårsaget af natriumkanalmutationer. Ugeskrift for læger. 2009;171(15):1261-5.

Author

Tfelt-Hansen, Jacob ; Olesen, Søren-Peter ; Svendsen, Jesper Hastrup ; Jespersen, Thomas. / Arvelige hjertesygdomme forårsaget af natriumkanalmutationer. In: Ugeskrift for læger. 2009 ; Vol. 171, No. 15. pp. 1261-5.

Bibtex

@article{95d56bd0359711df8ed1000ea68e967b,
title = "Arvelige hjertesygdomme for{\aa}rsaget af natriumkanalmutationer",
abstract = "Contraction of the heart is achieved through a delicately regulated conduction of electrical impulses. A pivotal element in the impulse propagation is the depolarising sodium current responsible for the initial depolarisation of the cardiomyocytes. Recent research has shown that mutations in the gene encoding the cardiac sodium channel (SCN5A) is associated with both rare forms of ventricular arrhythmia, and with the most frequent form of arrhythmia, atrial fibrillation. Udgivelsesdato: 2009-Apr-6",
author = "Jacob Tfelt-Hansen and S{\o}ren-Peter Olesen and Svendsen, {Jesper Hastrup} and Thomas Jespersen",
note = "Keywords: Atrial Fibrillation; Brugada Syndrome; Heart Conduction System; Humans; Long QT Syndrome; Muscle Proteins; Mutation; Sodium Channels",
year = "2009",
language = "Dansk",
volume = "171",
pages = "1261--5",
journal = "Ugeskrift for Laeger",
issn = "0041-5782",
publisher = "Almindelige Danske Laegeforening",
number = "15",

}

RIS

TY - JOUR

T1 - Arvelige hjertesygdomme forårsaget af natriumkanalmutationer

AU - Tfelt-Hansen, Jacob

AU - Olesen, Søren-Peter

AU - Svendsen, Jesper Hastrup

AU - Jespersen, Thomas

N1 - Keywords: Atrial Fibrillation; Brugada Syndrome; Heart Conduction System; Humans; Long QT Syndrome; Muscle Proteins; Mutation; Sodium Channels

PY - 2009

Y1 - 2009

N2 - Contraction of the heart is achieved through a delicately regulated conduction of electrical impulses. A pivotal element in the impulse propagation is the depolarising sodium current responsible for the initial depolarisation of the cardiomyocytes. Recent research has shown that mutations in the gene encoding the cardiac sodium channel (SCN5A) is associated with both rare forms of ventricular arrhythmia, and with the most frequent form of arrhythmia, atrial fibrillation. Udgivelsesdato: 2009-Apr-6

AB - Contraction of the heart is achieved through a delicately regulated conduction of electrical impulses. A pivotal element in the impulse propagation is the depolarising sodium current responsible for the initial depolarisation of the cardiomyocytes. Recent research has shown that mutations in the gene encoding the cardiac sodium channel (SCN5A) is associated with both rare forms of ventricular arrhythmia, and with the most frequent form of arrhythmia, atrial fibrillation. Udgivelsesdato: 2009-Apr-6

M3 - Tidsskriftartikel

C2 - 19416615

VL - 171

SP - 1261

EP - 1265

JO - Ugeskrift for Laeger

JF - Ugeskrift for Laeger

SN - 0041-5782

IS - 15

ER -

ID: 18763758