Thomas Jespersen

Thomas Jespersen

Professor

Member of:

    ORCID: 0000-0001-6575-360X

    Publication year:
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    171 - 180 out of 188Page size: 10
    1. 2004
    2. Published

      Basolateral localisation of KCNQ1 potassium channels in MDCK cells: molecular identification of an N-terminal targeting motif.

      Jespersen, Thomas, Rasmussen, Hanne Borger, Grunnet, Morten, Jensen, H. S., Angelo, K., Dupuis, D. S., Vogel, L. K., Jorgensen, N. K., Klærke, Dan Arne & Olesen, Søren-Peter, 2004, In: Journal of Cell Science. 117, Pt 19, p. 4517-26 9 p.

      Research output: Contribution to journal › Journal article › Research › peer-review

    3. Published

      Molecular determinants of voltage-gated sodium channel regulation by the Nedd4/Nedd4-like proteins.

      Rougier, J., van Bemmelen, M. X., Bruce, M. C., Jespersen, Thomas, Gavillet, B., Apothéloz, F., Cordonier, S., Staub, O., Rotin, D. & Abriel, H., 2004, In: American Journal of Physiology: Cell Physiology. 288, 3, p. C692-701

      Research output: Contribution to journal › Journal article › Research › peer-review

    4. 2003
    5. Published

      Mutational library analysis of selected amino acids in the receptor binding domain of envelope of Akv murine leukemia virus by conditionally replication competent bicistronic vectors

      Bahrami, S., Jespersen, T., Pedersen, F. S., Duch, M. R. & Jespersen, Thomas, 2 Oct 2003, In: Gene. 315, p. 51-61 11 p.

      Research output: Contribution to journal › Journal article › Research › peer-review

    6. Published

      Solid-phase synthesis and biological activity of a thioether analogue of conotoxin G1.

      Bondebjerg, J., Grunnet, Morten, Jespersen, Thomas & Meldal, Morten Peter, 2003, In: ChemBioChem. 4, 2-3, p. 186-94 8 p.

      Research output: Contribution to journal › Journal article › Research › peer-review

    7. Published

      Characterization of two new dominant ClC-1 channel mutations associated with myotonia.

      Grunnet, Morten, Jespersen, Thomas, Colding-Jørgensen, E., Schwartz, M., Klærke, Dan Arne, Vissing, John, Olesen, Søren-Peter & Dunø, M., 2003, In: Muscle & Nerve. 28, 6, p. 722-32 10 p.

      Research output: Contribution to journal › Journal article › Research › peer-review

    8. Published

      KCNE4 is an inhibitory subunit to Kv1.1 and Kv1.3 potassium channels.

      Grunnet, Morten, Rasmussen, H. B., Hay-Schmidt, Anders, Rosenstierne, M., Klærke, Dan Arne, Olesen, Søren-Peter & Jespersen, Thomas, 2003, In: Biophysical Journal. 85, 3, p. 1525-37 12 p.

      Research output: Contribution to journal › Journal article › Research › peer-review

    9. Published

      KCNQ1 channels sense small changes in cell volume.

      Grunnet, Morten, Jespersen, Thomas, MacAulay, Nanna, Jørgensen, N. K., Schmitt, Nicole, Pongs, O., Olesen, Søren-Peter & Klærke, Dan Arne, 2003, In: Journal of Physiology. 549, Pt 2, p. 419-27 8 p.

      Research output: Contribution to journal › Journal article › Research › peer-review

    10. 2002
    11. Published

      KCNE5 induces time- and voltage-dependent modulation of the KCNQ1 current.

      Angelo, K., Jespersen, Thomas, Grunnet, Morten, Nielsen, Morten Schak, Klærke, Dan Arne & Olesen, Søren-Peter, 2002, In: Biophysical Journal. 83, 4, p. 1997-2006 9 p.

      Research output: Contribution to journal › Journal article › Research › peer-review

    12. Published

      Activation of KCNQ5 channels stably expressed in HEK293 cells by BMS-204352.

      Dupuis, D. S., Schrøder, R. L., Jespersen, Thomas, Christensen, J. K., Christophersen, P., Jensen, B. S. & Olesen, Søren-Peter, 2002, In: European Journal of Pharmacology. 437, 3, p. 129-37 8 p.

      Research output: Contribution to journal › Journal article › Research › peer-review

    13. Published

      KCNE4 is an inhibitory subunit to the KCNQ1 channel.

      Grunnet, Morten, Jespersen, Thomas, Rasmussen, Hanne Borger, Ljungstrøm, T., Jorgensen, N. K., Olesen, Søren-Peter & Klærke, Dan Arne, 2002, In: Journal of Physiology. 542, Pt 1, p. 119-30 11 p.

      Research output: Contribution to journal › Journal article › Research › peer-review

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