Do the actions of glucagon-like peptide-1 on gastric emptying, appetite, and food intake involve release of amylin in humans?
Research output: Contribution to journal › Journal article › Research › peer-review
Amylin, cosecreted with insulin, has like glucagon-like peptide-1 (GLP-1) been reported to inhibit glucagon secretion, delay gastric emptying, and reduce appetite and food intake. We investigated whether the effects of GLP-1 on gastric emptying, appetite, and food intake are mediated directly or indirectly via release of amylin.
| Original language | English |
|---|---|
| Journal | The Journal of clinical endocrinology and metabolism |
| Volume | 95 |
| Issue number | 5 |
| Pages (from-to) | 2367-75 |
| Number of pages | 9 |
| DOIs | |
| Publication status | Published - 1 May 2010 |
- Adult, Amyloid, Appetite, Body Mass Index, C-Peptide, Cross-Over Studies, Diabetes Mellitus, Type 1, Gastric Emptying, Glucagon, Glucagon-Like Peptide 1, Hemoglobin A, Glycosylated, Humans, Hypoglycemic Agents, Insulin, Islet Amyloid Polypeptide, Reference Values, Single-Blind Method
Research areas
ID: 33940040