Arvelige hjertesygdomme forårsaget af natriumkanalmutationer
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Arvelige hjertesygdomme forårsaget af natriumkanalmutationer. / Tfelt-Hansen, Jacob; Olesen, Søren-Peter; Svendsen, Jesper Hastrup; Jespersen, Thomas.
In: Ugeskrift for læger, Vol. 171, No. 15, 2009, p. 1261-5.Research output: Contribution to journal › Journal article › Research › peer-review
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TY - JOUR
T1 - Arvelige hjertesygdomme forårsaget af natriumkanalmutationer
AU - Tfelt-Hansen, Jacob
AU - Olesen, Søren-Peter
AU - Svendsen, Jesper Hastrup
AU - Jespersen, Thomas
N1 - Keywords: Atrial Fibrillation; Brugada Syndrome; Heart Conduction System; Humans; Long QT Syndrome; Muscle Proteins; Mutation; Sodium Channels
PY - 2009
Y1 - 2009
N2 - Contraction of the heart is achieved through a delicately regulated conduction of electrical impulses. A pivotal element in the impulse propagation is the depolarising sodium current responsible for the initial depolarisation of the cardiomyocytes. Recent research has shown that mutations in the gene encoding the cardiac sodium channel (SCN5A) is associated with both rare forms of ventricular arrhythmia, and with the most frequent form of arrhythmia, atrial fibrillation. Udgivelsesdato: 2009-Apr-6
AB - Contraction of the heart is achieved through a delicately regulated conduction of electrical impulses. A pivotal element in the impulse propagation is the depolarising sodium current responsible for the initial depolarisation of the cardiomyocytes. Recent research has shown that mutations in the gene encoding the cardiac sodium channel (SCN5A) is associated with both rare forms of ventricular arrhythmia, and with the most frequent form of arrhythmia, atrial fibrillation. Udgivelsesdato: 2009-Apr-6
M3 - Tidsskriftartikel
C2 - 19416615
VL - 171
SP - 1261
EP - 1265
JO - Ugeskrift for Laeger
JF - Ugeskrift for Laeger
SN - 0041-5782
IS - 15
ER -
ID: 18763758