Sofia Hammami Bomholtz

Sofia Hammami Bomholtz

Postdoc

ORCID: 0000-0001-7221-4532

Publication year:
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11 - 20 out of 24Page size: 10
  1. 2016
  2. Published

    pH-dependent inhibition of K2P3.1 prolongs atrial refractoriness in whole hearts

    Skarsfeldt, M. A., Jepps, Thomas Andrew Qvistgaard, Bomholtz, Sofia Hammami, Abildgaard, L., Sørensen, U. S., Gregers, E., Svendsen, Jesper Hastrup, Diness, J. G., Grunnet, M., Schmitt, Nicole, Olesen, Søren-Peter & Bentzen, Bo Hjorth, Apr 2016, In: Pfluegers Archiv. 468, 4, p. 643-654 12 p.

    Research output: Contribution to journal › Journal article › Research › peer-review

  3. 2015
  4. Published

    Antiarrhythmic Mechanisms of SK Channel Inhibition in the Rat Atrium

    Skibsbye, L., Wang, X., Axelsen, L. N., Bomholtz, Sofia Hammami, Nielsen, Morten Schak, Grunnet, Morten, Bentzen, Bo Hjorth & Jespersen, Thomas, Aug 2015, In: Journal of Cardiovascular Pharmacology. 66, 2, p. 165-176 12 p.

    Research output: Contribution to journal › Journal article › Research › peer-review

  5. 2014
  6. Published

    Cell volume changes regulate Slick (Slo2.1), but not Slack (Slo2.2) K+ channels

    Tejada, M. D. L. A., Stople, K., Bomholtz, Sofia Hammami, Meinild, A., Poulsen, A. N. & Klærke, Dan Arne, 2014, In: P L o S One. 9, 10, p. 1-8 8 p., e110833.

    Research output: Contribution to journal › Journal article › Research › peer-review

  7. 2013
  8. Published

    A phosphoinositide 3-kinase (PI3K)-serum- and glucocorticoid-inducible kinase 1 (SGK1) pathway promotes Kv7.1 channel surface expression by inhibiting Nedd4-2 protein

    Andersen, M. N., Krzystanek, K., Petersen, F., Bomholtz, Sofia Hammami, Olesen, Søren-Peter, Abriel, H., Jespersen, Thomas & Rasmussen, Hanne Borger, 8 Nov 2013, In: The Journal of Biological Chemistry. 288, 52, p. 36841-54 14 p.

    Research output: Contribution to journal › Journal article › Research › peer-review

  9. Published

    Purinergic signalling - a possible mechanism for KCNQ1 channel response to cell volume challenges

    Bomholtz, Sofia Hammami, Willumsen, N. J., Meinild, A., Klærke, Dan Arne & Novak, Ivana, 2013, In: Acta Physiologica (Print). 207, 3, p. 503-515 13 p.

    Research output: Contribution to journal › Journal article › Research › peer-review

  10. 2012

  11. Evidence for a common pharmacological interaction site on K(Ca)2 channels providing both selective activation and selective inhibition of the human K(Ca)2.1 subtype

    Hougaard, C., Bomholtz, Sofia Hammami, Eriksen, B. L., Sørensen, U. S., Jensen, Marianne Lund, Strøbæk, D., Christophersen, P. & Bomholtz, Sofia Hammami, Feb 2012, In: Molecular Pharmacology. 81, 2, p. 210-9 10 p.

    Research output: Contribution to journal › Journal article › Research › peer-review

  12. 2011
  13. Submitted

    Upregulation of KCNQ1 currents by the KCNE1 beta subunit is not caused by insertion of KCNQ1 into the membrane

    Bomholtz, Sofia Hammami, Willumsen, N. & Klærke, Dan Arne, 2011, (Submitted). 1 p.

    Research output: Contribution to conference › Conference abstract for conference › Research

  14. 2010
  15. Published

    KCNE1-induced increase in KCNQ1 currents is not mediated through enhanced membrane expression

    Bomholtz, Sofia Hammami, Willumsen, N. J. & Klærke, Dan Arne, Apr 2010, In: The FASEB Journal. 24, 610.6

    Research output: Contribution to journal › Conference abstract in journal › Research

  16. Published

    KCNQ1 channel response to cell volume changes is not mediated by ATP release

    Bomholtz, Sofia Hammami, Willumsen, N., Novak, Ivana & Klærke, Dan Arne, 2010, In: Acta Physiologica Scandinavica. Supplementum. 198, Suppl 677, p. 213 1 p.

    Research output: Contribution to journal › Conference abstract in journal › Research

  17. Published

    Mechanism for strict regulation of certain K+ channels by small, fast changes in cell volume

    Tejada, M. D. L. A., Stolpe, K., Bomholtz, Sofia Hammami, Willumsen, N. J., Poulsen, A. N. & Klærke, Dan Arne, 2010, In: Biophysical Journal. Supp. 1, p. 534a

    Research output: Contribution to journal › Conference abstract in journal › Research

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