Loss of K+ currents in heart failure is accentuated in KChIP2 deficient mice

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Loss of K+ currents in heart failure is accentuated in KChIP2 deficient mice. / Grubb, Søren Jahn; Speerschneider, Tobias; Occhipinti, Dona; Fiset, Céline; Olesen, Søren-Peter; Thomsen, Morten Bækgaard; Callø, Kirstine.

I: Journal of cardiovascular electrophysiology, Bind 25, Nr. 8, 2014, s. 896-904.

Publikation: Bidrag til tidsskriftTidsskriftartikelForskningfagfællebedømt

Harvard

Grubb, SJ, Speerschneider, T, Occhipinti, D, Fiset, C, Olesen, S-P, Thomsen, MB & Callø, K 2014, 'Loss of K+ currents in heart failure is accentuated in KChIP2 deficient mice', Journal of cardiovascular electrophysiology, bind 25, nr. 8, s. 896-904. https://doi.org/10.1111/jce.12422

APA

Grubb, S. J., Speerschneider, T., Occhipinti, D., Fiset, C., Olesen, S-P., Thomsen, M. B., & Callø, K. (2014). Loss of K+ currents in heart failure is accentuated in KChIP2 deficient mice. Journal of cardiovascular electrophysiology, 25(8), 896-904. https://doi.org/10.1111/jce.12422

Vancouver

Grubb SJ, Speerschneider T, Occhipinti D, Fiset C, Olesen S-P, Thomsen MB o.a. Loss of K+ currents in heart failure is accentuated in KChIP2 deficient mice. Journal of cardiovascular electrophysiology. 2014;25(8):896-904. https://doi.org/10.1111/jce.12422

Author

Grubb, Søren Jahn ; Speerschneider, Tobias ; Occhipinti, Dona ; Fiset, Céline ; Olesen, Søren-Peter ; Thomsen, Morten Bækgaard ; Callø, Kirstine. / Loss of K+ currents in heart failure is accentuated in KChIP2 deficient mice. I: Journal of cardiovascular electrophysiology. 2014 ; Bind 25, Nr. 8. s. 896-904.

Bibtex

@article{6901728e052f43ebba877239643d7f3c,
title = "Loss of K+ currents in heart failure is accentuated in KChIP2 deficient mice",
abstract = "KV 4 together with KV Channel-Interacting Protein 2 (KChIP2) mediate the fast recovering transient outward potassium current (Ito,f ) in the heart. KChIP2 is downregulated in human heart failure (HF), potentially underlying the loss of Ito,f . We investigated remodeling associated with HF hypothesizing that KChIP2 plays a central role in the modulation of outward K(+) currents in HF.",
author = "Grubb, {S{\o}ren Jahn} and Tobias Speerschneider and Dona Occhipinti and C{\'e}line Fiset and S{\o}ren-Peter Olesen and Thomsen, {Morten B{\ae}kgaard} and Kirstine Call{\o}",
note = "This article is protected by copyright. All rights reserved.",
year = "2014",
doi = "10.1111/jce.12422",
language = "English",
volume = "25",
pages = "896--904",
journal = "Journal of Cardiovascular Electrophysiology",
issn = "1045-3873",
publisher = "Wiley-Blackwell",
number = "8",

}

RIS

TY - JOUR

T1 - Loss of K+ currents in heart failure is accentuated in KChIP2 deficient mice

AU - Grubb, Søren Jahn

AU - Speerschneider, Tobias

AU - Occhipinti, Dona

AU - Fiset, Céline

AU - Olesen, Søren-Peter

AU - Thomsen, Morten Bækgaard

AU - Callø, Kirstine

N1 - This article is protected by copyright. All rights reserved.

PY - 2014

Y1 - 2014

N2 - KV 4 together with KV Channel-Interacting Protein 2 (KChIP2) mediate the fast recovering transient outward potassium current (Ito,f ) in the heart. KChIP2 is downregulated in human heart failure (HF), potentially underlying the loss of Ito,f . We investigated remodeling associated with HF hypothesizing that KChIP2 plays a central role in the modulation of outward K(+) currents in HF.

AB - KV 4 together with KV Channel-Interacting Protein 2 (KChIP2) mediate the fast recovering transient outward potassium current (Ito,f ) in the heart. KChIP2 is downregulated in human heart failure (HF), potentially underlying the loss of Ito,f . We investigated remodeling associated with HF hypothesizing that KChIP2 plays a central role in the modulation of outward K(+) currents in HF.

U2 - 10.1111/jce.12422

DO - 10.1111/jce.12422

M3 - Journal article

VL - 25

SP - 896

EP - 904

JO - Journal of Cardiovascular Electrophysiology

JF - Journal of Cardiovascular Electrophysiology

SN - 1045-3873

IS - 8

ER -

ID: 106948937